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Therefore, although testing for Nnt WT expression in BL/6 mice with unknown or complex history of BL/6J and BL/6N intercrossing is important to assess their regulation of mitochondrial NADPH and E GSH , it will not guarantee that their glucose tolerance is as good as in BL/6N mice (if they express Nnt WT ), or as bad as in BL/6J mice (if they express Nnt Tr )

Animal models are valuable for identifying mechanisms and generating hypotheses, but they do not always translate directly to human outcomes

Phosphorylation of Mammalian Cytochrome c and Cytochrome c Oxidase in the Regulation of Cell Destiny: Respiration, Apoptosis, and Human Disease

This model proposes that mGSH depletion due to its covalent binding to NAPQI acts as a first-hit, which is followed by a burst of mitochondrial ROS production that activates a second hit involving mitogen-activated protein kinases (MAPKs) that in turn amplifies the wave of mitochondrial ROS generation (Lemasters, 1998)