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ROS may result in the dysfunctioning of OPCs through a number of cellular pathways, including the ERK1/2 and AMPK signalling cascades that cause an inactivation of the mTOR-P70S6K pathway, and hence negatively influence proliferation and differentiation of this cell type (Figure 2)
This band was not obvious in the free siRNA and in GNCnsRNA-treated tumours (Fig
Accordingly, both efferocytosis and -cyclodextrin have been investigated [182, 183]
