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Pharmaceutical agents such as Cox-2 inhibitors, thalidomide, megestrol, medroxyprogesterone acetate, melatonin, and DCA, and supplemental nutrients or antioxidants from food [35], such as -3 poly-unsaturated fatty acids, vitamin C, vitamin D, vitamin E, -hydroxy--methylbutyrate (HMB) [36] and lycopene, represent potential anti-cachexia treatments that are being explored in clinical trials

Though the mechanism remains unclear, these data suggest a relationship between peripheral apoE expression and inflammatory signaling that is isoform-dependent

First, identifying and targeting patients with an active inflammatory-fibrotic phenotype, typically characterized by elevated baseline IL-6 or high-sensitivity C-reactive protein, or by MRI-PDFF-evidenced steatosis with elevated T1 mapping values