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Importantly, the disease causing I148M variant was a gain of function for the interaction with ABHD5 and inhibition of lipolysis, and could promote TAG accumulation in an ABHD5-dependent manner (70)
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[DOI] [PMC free article] [PubMed] [Google Scholar] 58.Crown J., Sablin M.-P., Corts J., Bergh J., Im S.-A., Lu Y.S., Martnez N., Neven P., Lee K.S., Morales S., et al
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