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Ferroptosis inhibition improves respiratory function in Silicosis mice To determine whether inhibiting ferroptosis would delay Silicosis fibrosis in mice, we established a mouse Silicosis model and administered Fer-1 (Fig

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This intricate relationship highlights CHAC1 as a pivotal player in the cellular response to oxidative stress, linking metabolic and stress response pathways in a manner that can critically influence cell fate under stress conditions ( 4.2 Unfolded protein response CHAC1 plays a crucial role in the UPR, specifically within the ATF4-ATF3-CHOP signaling pathway