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found that the IKK S-glutathionylation by glutathione disulfide amplified TNF- mediated hepatotoxicity, revealing that IKK S -glutathionylation was the potential mechanism of NAFLD [57]

Therefore, the above studies suggest that vascular ROS promotes plaque stability by modulating the expression and activities of MMPs

Interestingly, it is found that GSTP1 expression is regulated by the Keap1-Nrf2-ARE pathway and GSTP1 catalyzes the S-glutathionylation of cysteine residues of Keap1 protein, while S-glutathionylation of Keap1 leads to Nrf2 activation and increases the expression of GSTP1 (Ikeda et al., 2002