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Although the exact mechanisms by which the coordinated actions of GLP-1 receptor agonism and NMDA receptor antagonism correct metabolic diseases await further elucidation, the extensive alterations in transcriptomic and proteomic responses within the hypothalamus, linked to glutamatergic signalling and synaptic plasticity, imply that the conjugate might elicit neurostructural changes in GLP-1-receptor-expressing neurons

Mechanisms, clinical trial results, side effect profiles, dosing protocols, regulatory status, and practical considerations for anyone trying to understand which compound might better align with their goals

GLP-1 medications may increase heat-related risks by suppressing thirst and reducing fluid intake

The HPA axis mainly includes the parvocellular corticotropin-releasing hormone (CRH), argininevasopressin (AVP) neurons of the hypothalamic paraventricular nuclei (PVN), anterior pituitary corticotrophs and adrenal cortisol-producing cells