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Description
When it binds to GLP-1 receptors in the brain and digestive system, it slows gastric emptying, reduces appetite signaling, and improves insulin sensitivity

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By decreasing -cell activity and triggering the nuclear factor kappa-light-chain-enhancer of activated cells (NF-) transcription factor, which damages and kills -cells, IL-1 contributes to the pancreatic autoimmune response
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