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[5] This rapid breakdown explains why therapeutic GLP-1 receptor agonists have been chemically modified to resist DPP-4 degradation, thereby extending their duration of action and clinical efficacy in managing diabetes and obesity
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Figure 4 indicates that compared with other hypoglycemic agents in AERSMine, PRR was increased when GLP-1RA was combined with DPP4i (pancreatic neoplasms malignant: GLP-1RA vs
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