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Regarding a potential explanation to the discrepancy in food intake reductions between studies, glucagon efficacy on satiety seems in part dependent on the state of ARC-localized CaMKK sensitivity within a GcgR/PKA/CaMKK/AMPK/AgRP axis, of which the obese state may negatively modulate [3]
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From the clinical perspective, GLP-1R gene polymorphism is associated with AUD, and exenatide moderately reduces alcohol drinking in overweight AUD patients
Mills KT, et al
