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They observed that the iGLP1-E2 conjugate prevented STZ-induced insulin-deficient diabetes, thereby demonstrating that, in vivo, the inactive GLP-1 was able to bind the GLP-1R and to direct E2 to -cells for protection
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The results illuminated exceptional RT-associated antitumor immune responses triggered by the synergistic therapeutic strategy of aCTLA-4 and IL-12
Maltose-Binding Protein (MBP), a Secretion-Enhancing Tag for Mammalian Protein Expression Systems
